Uric acid, a natural byproduct resulting from the breakdown of purines, constitutes a normal metabolic waste in the body. Purines, present both endogenously and in foods such as liver, shellfish, and alcohol, contribute to its formation. Ordinarily, the body eliminates uric acid through urine or bowel movements. However, an excess may occur due to factors like overproduction, compromised kidney function, or influences such as high-purine food intake or specific medications (e.g., diuretics, aspirin, niacin). Elevated uric acid levels can precipitate crystal formation, leading to the painful inflammatory condition known as gout and contributing to the development of kidney stones. Vigilant monitoring of uric acid levels and the adoption of lifestyle adjustments are pivotal for effectively managing and preventing complications associated with heightened uric acid concentrations in the bloodstream.
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